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Graduate Student Researcher
Department of Molecular, Cellular, and Integrative Physiology, UCLA
E-mail: apeter@ucla.edu

Principle Research Interests
Muscular dystrophies are inherited genetic disorders characterized by progressive muscle wasting and weakness. Dystrophic muscle undergoes repeated cycles of degeneration and regeneration, which is characterized by the presence of hypertrophic fibers with central nuclei. Akt, also known as protein kinase B (PKB), is a serine-threonine kinase that has been shown to modulate a wide range of cellular processes including apoptosis and translation efficiency. I am currently investigating the activation of Akt associated pathways in various animal models of muscular dystrophy. I hypothesize that skeletal muscle hypertrophy and atrophy in muscular dystrophies are controlled by perturbations in Akt phosphorylation. I will also examine whether direct manipulation of the Akt/PKB pathway counteracts muscle wasting in muscular dystrophy.
Education
Ph.D.
  • University of California, Los Angeles, CA 90095
    Interdepartmental Program in Molecular, Cellular and Integrative Physiology
2003 - present
B.S.
  • Colorado State University, Fort Collins, CO 80523
    Department of Microbiology, Immunology, and Pathology
1999-2003
Positions Held
  • Undergraduate Teaching Assistant 2002 Dept. Microbiology
Selected Presentations
2003
Peter, A., Lambert, M., McNeil, M. Testing for Protein-Protein Interactions with the Mycobacterium smegmatis EmbB Transmembrane Protein. Undergraduate Research and Creative Symposium. Colorado State University. April 2003
2001
Richards (Peter), A., Jones, V., Scherman, M., McNeil, M. Drug Screening Assay: Optimization of the Enzymatic Formation of TDP-rhamnose Necessary for Mycobacterium tuberculosis Cell Wall Formation. Undergraduate Research and Creative Symposium. Colorado State University. April 2001
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